Acute & General Medicine, 21- 22 November 2017, EXCEL LONDON

EUROPE’S MUST ATTEND CLINICAL TRAINING
CONFERENCE FOR ALL HOSPITAL DOCTORS 

12-13 NOVEMBER 2019, EXCEL LONDON

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EUROPEAN MEDICAL ASSOCIATION
ANNUAL CONFERENCE

HOSTING THE EUROPEAN ACUTE CARE CONFERENCE

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2018 Seminar Programme

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Inotropes and vasoactive agents

Streams: Critical Care
Cardiovascular drug actions include chronotropy (firing of sinoatrial node), inotropy (contractility), lusitropy (myocardial relaxation), and dromotropy (conduction velocity of AV node).
Vasoactive Agents can broadly be classified as:
Inotropes that increase myocardial contractility, vasopressors that cause vasoconstriction leading to increase in systemic/pulmonary vascular resistance; inodilators that promote contraction ability that also cause decrease in systemic/pulmonary vascular resistance.
No inotropic agents have been shown to have superiority over any others in quality trials.
The goal of intervention is to reverse the state of inadequate tissue perfusion that results in life-threatening impairment of oxygen and nutrient delivery.
Learning outcomes:
  1. Understanding and identifying the underlying pathogenesis of shock is essential to steer the pace and degree of haemodynamic intervention.
  2. Selection of vasoactive agents should be based on goal-directed correction of shock, with frequent assessments of the dynamic demand-supply match of macro- and micro-circulation.
  3. Non-catecholamine-based inotropes and vasoactive agents include calcium, milrinone (phosphodiesterase III inhibitor), levosimendan (calcium-channel sensitizer)
Speakers
Dr Amy Chan-Dominy, Cardiothoracic & Paediatric Intensivist - Royal Brompton Hospital, London

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